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There are several concerns in this field: firstly, despite the association of the mitochondria-derived peptide MOTS-c with bone metabolism and current studies confirming the effects of MOTS-c intervention, the role and mechanism of endogenous skeletal MOTS-c in regulating bone metabolism have not been evaluated

For comparisons among multiple groups, one-way ANOVA followed by Tukeys post hoc test was applied to assess all possible pairwise differences

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These suggests that the neuroprotective effects of berberine may be related to an excitatory mechanism that promotes cell survival and antioxidant-related signaling pathways

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